MITOCHONDRIAL RESPIRATION

Mitochondrial respiration is the secret to disease.

Despite an ever expanding industrial medical complex, the global burden of chronic disease continues to expand. The BX Energy Catalyst™ represents novel weaponry against disease. The catalyst’s mode of action is aimed at restoring mitochondrial respiration through dehydrogenation of bacterial proteins and other toxins blocking the metabolism of fuels. This brief study identifies the metabolic pathways involved and establishes the viability of the BX Protocol™ in correcting mitochondrial bioelectric deficiencies. The intracellular organelle known as the mitochondrion is responsible for providing most of the cellular ATP, a complex chemical molecule critical in sustaining life.

The slightest disturbance in mitochondrial respiration leads to a variety of pathological conditions, such as:

  • Cancer
  • Diabetes
  • Multiple sclerosis
  • Parkinson’s
  • Congestive heart failure

We propose that defects in mitochondrial respiration are a fundamental cause leading to a host of chronic diseases. A key component of mitochondrial respiration relates to the function of a newly identified unsaturated protein often associated with cell wall deficient organisms. We will discuss some fundamental molecular details concerning this protein (K-85) and its role in the etiology and pathogenesis of numerous conditions.

We claim that “Stealth pathogens”, represented as filterable pleomorphic variants of a variety of bacterial species, are in fact the primary cause of many chronic pathologies. The organism can be cultured from isolated mitochondria and is directly responsible for NADH-coenzyme Q reductase deficiencies and the resulting impairment of the oxidative mechanisms of the cell. They are characterized by:

  • Weakness
  • Exercise intolerance
  • Fluctuating acidity profile
  • Decline in mitochondrial respiratory rates.

Simply put, the toxic proteins associated with stealth pathogens combine with chemical groups in complex I, preventing oxygen from accepting electrons. They trigger transmembrane proteins that prematurely dissipate the proton gradient and drain the electrical potentials required for energy production.

In this manner, the bacterial toxins could be seen as causing an electrical short circuit where the batteries (mitochondria) lose their charge and are no longer able to produce energy.

Delta’s investigation of the relationship between mitochondrial respiration and stealth pathogens has been ongoing for the past eighteen years and has proven instrumental in establishing the fundamental mechanisms of impairment pertaining to several conditions.

To learn more about mitochondria and the BX Protocol™, watch this informational video.

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